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Cerebral Palsy

  • Non-progressive upper motor neuron disease (static encephalopathy) due to injury to immature brain

Epidemiology

  • Onset is before first two years of life
  • Most common cause of chronic childhood disability
  • Slightly more common in males
  • Higher incidence in low income countries

Etiology

  • Antenatal factors
    • Prematurity
    • Maternal infection –  TORCH
    • Neonatal vascular incidents – emboli from placental thrombosis
  • Perinatal factors
    • Birth asphyxia due to instrumental delivery
    • Non-vertex presentation
    • Placental abruption
  • Post natal factors
    • Hyperbilirubinemia
    • Neonatal sepsis
    • Respiratory distress
    • Head injury – child abuse, shaken baby syndrome

Types

Spastic (most common)

Ataxic (least common)

  • Caused by damage to cerebellar structures – responsible for coordinating muscle movements and balance
  • Clinical features
    • Problems in coordination – especially in arms, legs, and trunk
    • Decreased muscle tone
    • Intention (action) tremor – occurs when carrying out a specific, deliberate movement e.g. tying shoe laces, writing etc
      • Tremor intensifies as the hand gets closer to accomplishing the intended task

Dyskinetic

  • Caused by damage to the basal ganglia and the substantia nigra – e.g. in bilirubin encephalopathy and hypoxic-ischemic brain injury  
  • Extrapyramidal form of cerebral palsy – divided into two groups
    • Choreoathetosis – characterised by involuntary movements
    • Dystonia – characterized by slow, strong contractions; can occur locally or encompass the whole body
  • Clinical diagnosis of usually occurs within 18 months of birth – based on motor function and neuroimaging

Classification – Gross Motor Function Classification Scale (GMFCS)

  • Level I – near normal gross motor function, independent ambulator
  • Level II – walks independently, but with difficulty on uneven surfaces, minimal ability to jump
  • Level III – walks with assistive devices
  • Level IV – severely limited walking ability, mainly uses wheelchair
  • Level V – nonambulator, dependent in all aspects of care

Orthopaedic clinical features

  • Contractures
  • Fractures
  • Upper extremity deformities
    • Shoulder internal rotation contracture
    • Forearm pronation deformity
    • Wrist flexion deformity
    • Thumb in palm deformity
  • Hip subluxation and dislocation 
  • Spinal deformity
    • Scoliosis – more likely to progress than idiopathic scoliosis
  • Foot deformities
    • Equinus
    • Hallux valgus
    • Equinoplanovalgus
    • Equinocavovarus
  • Gait disorders
    • Equinus gait
    • Jump gait
    • Crouch gait
    • Stiff knee gait

Diagnosis

  • Clinical and Perinatal history
  • Physical exam – general musculoskeletal exam, gait, spine, hip, foot, ankle
  • X-ray
  • MRI – shows changes in the brain
    • Periventriuclar leukomalacia white matter lesions (most common)
    • Brain malformations

Treatment

Non-operative

  • Physiotherapy
  • Bracing
  • Medication for spasticity
    • Botulinum A – competitive inhibitor of presynaptic cholinergic receptors
    • Baclofen – GABA agonist, reduces muscle tone

Operative

  • Soft tissue releases – to improve function in a child (3-5 years old) with spasticity
    • Tenotomy, tendon lengthening, tendon transfer
  • Selective dorsal rhizotomy – neurosurgical resection of the nerve rootlets in the spinal cord that are sending abnormal signals to the muscles. Corrects muscle spasticity
  • SEMLS surgery – Single Event, Multi-level Surgery
    • To limit multiple surgeries and decreased rehabilitation time
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